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Tip: To review an author’s works published in PEP-Web…

PEP-Web Tip of the Day

The Author Section is a useful way to review an author’s works published in PEP-Web. It is ordered alphabetically by the Author’s surname. After clicking the matching letter, search for the author’s full name.

For the complete list of tips, see PEP-Web Tips on the PEP-Web support page.

Carter, C.S. (2017). The Role of Oxytocin and Vasopressin in Attachment. Psychodyn. Psych., 45(4):499-517.

(2017). Psychodynamic Psychiatry, 45(4):499-517

The Role of Oxytocin and Vasopressin in Attachment

C. Sue Carter, Ph.D.

Selective relationships and attachments are central to human health and well-being, both in current societies and during the course of evolution. The presence or absence of social bonds has consequences across the lifespan. The neurobiology of attachment is grounded in neuroendocrine substrates that are shared with reproduction and survival. Experimental studies of species, such as sheep or prairie voles, capable of showing selective social behaviors toward offspring or partners, have provided empirical evidence for the role of oxytocin and vasopressin in the formation of selective attachments. Developmental exposure to social experiences and to peptides, including oxytocin and vasopressin, also can “retune” the nervous system, altering thresholds for sociality, emotion regulation, and aggression. Without oxytocin and without the ability to form attachments the human brain as we know it could not exist. Knowledge of the neurobiology of attachment, and especially the role of oxytocin, also has implications for understanding both healthy behavior and treating mental disorders.

[This is a summary or excerpt from the full text of the article. PEP-Web provides full-text search of the complete articles for current and archive content, but only the abstracts are displayed for current content, due to contractual obligations with the journal publishers. For details on how to read the full text of 2017 and more current articles see the publishers official website.]

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